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He created the ‘Critically Ill Airway’ course and teaches on numerous courses around the world. He coordinates the Alfred ICU’s education and simulation programmes and runs the unit’s education website, INTENSIVE.

He is actively involved in in using translational simulation to improve patient care and the design of processes and systems at Alfred Health.

FACEBOOK CRITICAL OPS ALPHA PROFESSIONAL

He has completed fellowship training in both intensive care medicine and emergency medicine, as well as post-graduate training in biochemistry, clinical toxicology, clinical epidemiology, and health professional education. He is an internationally recognised Clinician Educator with a passion for helping clinicians learn and for improving the clinical performance of individuals and collectives.Īfter finishing his medical degree at the University of Auckland, he continued post-graduate training in New Zealand as well as Australia’s Northern Territory, Perth and Melbourne. He is on the Board of Directors for the Intensive Care Foundation and is a First Part Examiner for the College of Intensive Care Medicine. He is also a Clinical Adjunct Associate Professor at Monash University. He is a co-founder of the Australia and New Zealand Clinician Educator Network (ANZCEN) and is the Lead for the ANZCEN Clinician Educator Incubator programme. -> use of noradrenaline strongly related (p Splanchnic function not worsened by NAĬhris is an Intensivist and ECMO specialist at the Alfred ICU in Melbourne.low dose dopamine then randomized to high dose dopamine vs noradrenaline.effect of norepinephrine on the outcome of septic shock.-> criticisms = underpowered, methodology suboptimal, strict inclusion criteria.-> no significant difference: mortality, BP, time to haemodynamic stability, duration of vasopressor therapy, time to organ dysfunction resolution, adverse effects.noradrenaline vs adrenaline in Septic Shock.-> adrenaline – transient lactic acidosis, hyperglycaemia, tachycardia.-> no significant difference in mortality, LOS, ventilation, shock duration.Noradrenaline vs Adrenaline to treat hypotension (sepsis or cardiogenic failure).-> no significant difference in mortality 28 days.Patients on low dose Norad randomised to Vasopressin vs Norad.-> no significant evidence to say that one is better than the other.-> dopamine: increased risk of arrhythmias.

6 different vasopressors (alone or in combination with dobutamine or dopexamine).Elimination – short t1/2, unchanged in urine.increased afterload and beta effects may increased myocardial work and oxygen consumption.hypotension refractory to fluid resuscitation (primarily distributive shock such as septic shock, neurogenic shock, post-bypass vasoplegia and drug-induced).0.1 to 1 mcg/kg/min IV via central line.

clear, colorless solution, 2mg/mL, norepinephrine bitartrate.

increased venoconstriction -> increased venous return -> increased preload increased SVR -> increased afterload increased DBP and coronary perfusion pressure

direct alpha and beta adrenergic receptor agonist (vasopressor and weak inotropes).

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